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Jan A. Moynihan, Ph.D.
Dr. Jan A. Moynihan is an Associate Professor in the Departments of Psychiatry, Microbiology and Immunology, and Oncology at the University of Rochester Medical Center, Rochester, New York. Dr. Moynihan received her B.A. degree in Biology from the University of Rochester and did graduate study in Integrative Biology at Washington University, St. Louis, Missouri. She received an M.S. degree and was awarded her Ph.D. in Microbiology/Immunology from the University of Rochester. Dr. Moynihan's clinical, scholarly, and teaching interests lie in psychoneuroimmunology. She is a member of the American Association of Immunologists and the Psychoneuroimmunology Research Society. Currently, she is the Principal Investigator (PI) on a project funded by the Public Health Service (PHS), studying the effects of stress in mice that are immunocompromised and either skin grafted or infected with virus. She also is the PI on another PHS-funded grant that investigates the effects of endogenous opioids produced by stressed mice on type 1 and type 2 cytokine production and immune effector mechanisms.
Selected Recent Publications:
Callahan TA, Moynihan JA. The effects of chemical sympathectomy on T cell cytokine responses are not mediated by altered peritoneal exudate cell function or an inflammatory response. Brain, Behav Immun 2002;16:33-45.
Kohut M, Boehm G, Moynihan JA. Prolonged exercise suppresses antigen-specific cytokine response to upper respiratory infection. J Applied Physiol 2001;90:678-684.
Larson MR, Duberstein PR, Talbot N, Caldwell C, Moynihan JA. Preliminary examination of a pre-surgical psychosocial intervention for breast cancer patients: psychological distress and the immune response. J Psychosomatic Res 2000;48:187-194.
Lyness JM, Moynihan JA, Williford DJ, Cox C, Caine ED. Depression, medical illness, and interleukin-1b in older cardiac patients: a pilot study testing a model of depressive pathogenesis. Int J Psychiatry Med 2001;31:321-326.
Moynihan JA, Karp JD, Cohen N, Ader R. Immune deviation following stress odor exposure: role of endogenous opioids. J Neuroimmunol 2000;102:145-153.
Papathanassoglou ED, Moynihan JA, Ackerman MH, Mantzoros CS. Serum leptin levels are higher but are not independently associated with severity or mortality in the multiple organ dysfunction/systemic inflammatory response syndrome: a matched case control and a longitudinal study. Clin Endocrinol 2001;54:225-33.
Papathanassoglou ED, Moynihan JA, Vermillion DL, McDermott MP, Ackerman MH. Soluble fas levels correlate with multiple organ dysfunction severity, survival and nitrate levels, but not with cellular apoptotic markers in critically ill patients. Shock 2000;14:107-12.
Rice PA, Boehm GW, Moynihan JA, Bellinger DL, Stevens SY. Chemical sympathectomy increases the innate immune response and decreases the specific immune response in the spleen to infection with Listeria monocytogenes. J Neuroimmunol 2001;114:19-27.
Rice PA, Boehm GW, Moynihan JA, Bellinger DL, Stevens SY. 2002. Chemical sympathectomy alters numbers of splenic and peritoneal leukocytes. Brain, Behav Immun 2002;16:62-73.
March 2002
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